Aerosol Inhalation: Recent Research Frontiers: Proceedings by Morton Lippmann Ph.D. (auth.), J. C. M. Marijnissen, L.

By Morton Lippmann Ph.D. (auth.), J. C. M. Marijnissen, L. Gradoń (eds.)

A staff of hugely esteemed aerosol scientists - physicists, chemists, biologists and toxicologists - met in Warsaw in September, 1995, for you to speak about the most recent advancements within the box of aerosol healthiness topics, together with inhalation, lung delivery and deposition. the connection of those topics with the surroundings used to be additionally addressed.
This ebook includes the complaints of the Workshop and likewise records the consequent panel discussions.
topic Index included.

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Extra info for Aerosol Inhalation: Recent Research Frontiers: Proceedings of the International Workshop on Aerosol Inhalation, Lung Transport, Deposition and the Relation to the Environment: Recent Research Frontiers, Warsaw, Poland, September 14–16, 1995

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IARC periodically brings together knowledgeable scientists from around the world as a panel to review the available scientific information on specific agents or exposure circumstances for the purpose of classifying the agents and exposure situations as to the carcinogeniC potential for humans. In the past, the review process has ·focused on consideration of three types of data: (1) studies of cancer in humans, (2) studies of cancer in laboratory animals, and (3) other data relevant to evaluation of carcinogenicity, which are frequently referred to as supporting data.

Thus the number of particles deposited and their surface area would be quite low as compared with similar lung burdens of carbon black or diesel exhaust soot particles, which are much smaller in size and also have a higher specific surface area. An understanding of the specific mechanisms involved in induction of lung tumors in rats is crucial in deciding how these data can be used for assessing human lung cancer risk for exposure to these agents (McClellan, 1996; Driscoll, 1996; Watson and Valberg, 1996).

Risk factors: particles? . ,~ ~ mechanism or (b) specific genotoxic mechanism. particles producing lung cancer via (a) nonspecific inflammation-mediated Figure 9. J ::l s:::: 0) ::l E o U) - t ~ Noninflammatory lung particle dose mutations, and lung tumors. The other curve has a shallow slope at low levels of exposure since the toxicant is assumed to have some inherent genotoxicity that is manifest even in the absence of inflammation. The implications of these two alternative exposure-response relationships will be addressed in the next section of this article.

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